Publications
1. Iadecola C, Park L and Capone C: Princeton Conference: Threats to the mind: aging, amyloid and hypertension. Stroke, 40:S40-S44, 2009. 2. Park L, Zhou P, Pitstick R, Capone C, Anrather J, Norris EH, Younkin L, Younkin S, Carlson G, McEwen BS, & Iadecola C. Nox2-derived radicals contribute to neurovascular and behavioral dysfunction in mice overexpressing the amyloid precursor protein. Proc Natl Acad Sci USA, 105:1347-1352, 2008. 3. Park L, Gallo EF, Anrather J, Wang G, Norris EH, Paul J, Strickland S, & Iadecola C. Key role of tissue plasminogen activator in neurovascular coupling. Proc Natl Acad Sci USA, 105:1073-1078, 2008. 4. Kunz A, Park L, Abe T, Gallo E, Anrather J, Zhou P, & Iadecola C. Neurovascular protection by ischemic tolerance: role of nitric oxide and reactive oxygen species. J. Neurosci, 27:7083-7093, 2007. 5. Park L, Anrather J, Girouard H, Zhou P, & Iadecola C. Nox2-derived reactive oxygen species mediate neurovascular dysregulation in the aging mouse brain. J. Cereb. Blood Flow Metab, 27:1908-1918, 2007. 6. Girouard H, Park L, Anrather J, Zhou P, & Iadecola C. Cerebrovascular Nitrosative Stress Mediates Neurovascular and Endothelial Dysfunction Induced by Angiotensin II. Arterioscler. Thromb. Vasc. Biol. 27:303-309, 2007. 7. Kawano T, Anrather J, Zhou P, Park L, Wang G, Frys KA, Kunz A, Cho S, Orio M, & Iadecola C. Prostaglandin E2 EP1 receptors: downstream effectors of COX-2 neurotoxicity. Nat. Med. 12:225-229, 2006. 8. Girouard H, Park L, Anrather J, Zhou P, & Iadecola C. Angiotensin II attenuates endothelium-dependent responses in the cerebral microcirculation through nox-2-derived radicals. Arterioscler. Thromb. Vasc. Biol. 26:826-832, 2006. 9. Cho S, Park EM, Febbraio M, Anrather J, Park L, Racchumi G, Silverstein RL, & Iadecola C. The class B scavenger receptor CD36 mediates free radical production and tissue injury in cerebral ischemia. J. Neurosci. 25:2504-2512, 2005. 10. Park L, Anrather J, Zhou P, Frys K, Pitstick R, Younkin S, Carlson GA, & Iadecola C. NADPH oxidase-derived reactive oxygen species mediate the cerebrovascular dysfunction induced by the ß-amyloid peptide. J Neurosci, 25:1969-1977, 2005. 11. Park L, Anrather J, Zhou P, Frys K, Wang G, & Iadecola C. Exogenous NADPH increases cerebral blood flow through NADPH oxidase-dependent and -independent mechanisms. Arterioscler. Thromb. Vasc. Biol. 24:1860-1865, 2004. 12. Park L, Anrather J, Forster C, Kazama K, Carlson GA, & Iadecola C. Ab-induced vascular oxidative stress and attenuation of functional hyperemia in mouse somatosensory cortex. J. Cereb. Blood Flow Metab. 24:334-342, 2004.
